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Viral Hemorrhagic Septicemia Virus genetic diversity
among invasive and native reservoir fishes of the
Thousand Islands region, St. Lawrence River
John M. Farrel l ( jmfarrel l@esf.edu)
Anna L. Haws (alconkly@syr .edu)
Rodman G. Getchel l ( rgg4@cornel l .edu)
hn M. Farrel l ( jmfarrel l@esf.edu)
Anna L. Haws (alconkly@esf.edu)
Rodman G. Getchel l (RGG4 @esf.edu)
PREPARED BY:
John M. Farrell
Professor/Director, Thousand Islands Biological Station
SUNY College of Environmental Science and Forestry,
Syracuse, NY
Anna L. Haws (Conklyn)
Ph.D. Candidate
SUNY College of Environmental Science and Forestry,
Syracuse, NY
Rodman G. Getchell
Assistant Research Professor, Aquatic Animal Health Program
Cornell University College of Veterinary Medicine,
Ithaca, NY
PREPARED BY:
John M. Farrell
Professor/Director, Thousand Islands Biological Station
SUNY College of Environmental Science and Forestry,
This report was prepared for the New York State Water Resources Institute (NYSWRI) and the NYS
DEC Bureau of Invasive Species and Ecosystem Health with support from the NYS Environmental Protection Fund
2022
\.\. U_N/ v~
l ~~
""' to 0 lb
,tl>ED 1'·0•
mailto:jmfarrell@esf.edu
mailto:alconkly@syr.edu
mailto:jmfarrell@esf.edu
mailto:alconkly@esf.edu
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› 02
Abstract and Summary
› 02
Introduction
› 02
Methods
› 02
Results and Discussion
› 02
Policy Implications
› 02
Student Training
› 02
References
› 02
Appendices
Abstract and Summary…………………………………………………………………………………..…………3
Policy Implications……………….……………………………………………………………………….…………3
Introduction………………………………………………………………………………………………….…………4
Methods……………………………………………………………………………….………………………………….6
Results and Discussion……………………………………………………………………………………………10
Student Training………………………………………………………………………………………………….….19
References……………………………………………………………………………………..……………………….19
Appendices…………………………………………………………………………………………………………….20
Contents
VHSV-positive muskellunge mortality recovered from the St. Lawrence River near Waddington, NY
during April 2022. This specimen was sequenced as part of this study. collected for this study.
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Viral Hemorrhagic Septicemia Virus (VHSV), is a World
Organization for Animal Health (OIE) reportable fish
pathogen found across the northern hemisphere, and lineage
VHSV-IVb was first detected in the Great Lakes basin
following sizable fish kills in the mid-2000’s. The biological
and environmental factors that promote VHSV persistence
following establishment are not well understood. Therefore,
this study is investigating annual, spatial, and host species
patterns of viral genetic structure in the upper St. Lawrence
River (USLR), an ecosystem where VHSV has persisted for 20
years. The USLR supports large populations of invasive
round gobies, and these fish are critical to provide an
environment suitable to rapid viral reproduction, evolution
of new viral isolates, and dissemination of those isolates to
the native community.
Keywords
Invasive Species, Pathogen, Conservation
Policy Implications
Foremost, this study contributes to assessing
environmental considerations for a long-term sportfish
restoration program targeted at identifying and protecting
muskellunge spawning and nursery sites in the St.
Lawrence River. This research also contributes to the
greater knowledge regarding VHSV in the Laurentian Great
Lakes watershed and provides additional evolutionary
context to evaluate the epidemiology of this fish pathogen.
Finally, this study represents the final chapter in Mrs.
Haws’ dissertation to achieve her Doctor of Philosophy
degree in Fish & Wildlife Biology and Management from the
SUNY College of Environmental Science and Forestry.
Executive
Summary
Summary
Points of
Interest
1. Invasive round gobies harbor VHSV,
and are essential for the
amplification, spread, and evolution
of the virus in the St. Lawrence
River. Current VHSV sequence
types have caused mortality in the
valuable muskellunge population.
2. The genetic composition of VHSV
that exists in the St. Lawrence River
is substantially diverged from the
original reference strain, illustrating
this virus’ capacity to exploit
mutation to persist in the
environment.
3. Genetic haplotypes are capable of
sustaining infections for extended
periods of time and dispersing
throughout the St. Lawrence River.
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Introduction
Piscine novirhabdovirus, or Viral
Hemorrhagic Septicemia Virus (VHSV), is a World
Organization for Animal Health (OIE) reportable
fish pathogen found in marine, estuarine, and
freshwater ecosystems across the northern
hemisphere (OIE, 2019). A novel lineage, VHSV-
IVb, was detected in the Great Lakes basin
following sizable fish kills in the mid-2000’s.
Through IVb’s history in the watershed,
researchers have documented more than 30 fish
species vulnerable to infection, high rates of
genetic mutation, recurrent outbreaks, and
expanding geographic range (Getchell et al.,
2019). While this pathogen is extremely generalist
in its capacity to infect many species, sensitivity
to infection is highly variable between fishes, and
the biological and environmental factors that
influence VHSV persistence following
establishment are not well understood (Kim &
Faisal, 2010).
This research is investigating biotic and
abiotic patterns of viral genetic diversity in the
upper St. Lawrence River (USLR), an ecosystem
where the virus has persisted for 20 years. The
emergence of VHSV in this region coincided with
dramatic declines of spotted muskellunge, an
apex predatory fish in the SLR, making VHSV
research critical to conservation efforts. Previous
investigations in the USLR have emphasized
consistently high prevalence in surveys of the
virus, frequently observed fish kills since 2006
and enduringly suppressed populations of
muskellunge (Cornwell et al. 2014, Farrell et al.
2017, Getchell et al. 2019). One dominant threat to
their population recovery is invasion of nursery
bays by large abundances of round gobies
(Neogobius melanostomus). These invasive fish
harbor VHSV and are considered essential to
create an environment suitable to rapid viral
reproduction, evolution of new viral isolates, and
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dissemination of
those isolates to the native community (Cornwell
et al. 2012; Cornwell et al. 2014; Eckerlin et al. 2011;
Getchell et al. 2019; Farrell et al. 2017; Stepien &
Niner 2020).
In-depth evolutionary analyses can
improve ecosystem managers’ prediction
capacity for transmission patterns, relative
virulence, and host responses toward pathogens
(Stepien et al., 2015). RNA viruses experience
rapid genomic changes through their small
genomes, lack of polymerase proofreading, and
short generation times (Stepien et al., 2015).
Genetic variation is expected to permit VHSV to
further adapt to new environments and host
species, as well as to evade host immune
responses and facilitate long time maintenance in
populations (Getchell et al., 2017; Stepien et al.,
2015). Sequencing multiple genes is necessary to
identify genetic changes that can be reflected in
epidemiological behavior, and it’s been shown
that just a few amino acid changes can result in
altered VHSV virulence (Al-Hussinee et al., 2011;
Getchell et al., 2017). Continual monitoring of
VHSV types in fish populations across its range is
fundamental to acknowledge changes to the virus’
genetic population structure and the relation to
outbreak frequency and overall persistence
(Garver et al., 2013).
Previous investigations have shown that
VHSV-IVb has diversified extensively following its
first appearance in the Great Lakes (Cornwell et
al. 2014; Thompson et al. 2011; Stepien et al. 2015).
Genetic diversity analyses have largely used
nucleotide sequences of the nucleoprotein (N)
and glycoprotein (G) genes (Cornwell et al., 2012;
Dadar, 2020). The N gene is the most abundantly
transcribed genomic region, and the protein
product closely associates with genomic RNA and
facilitates interaction with viral polymerase
(Kurath & Winton, 2008). Chimeric VHSV
experiments have demonstrated that the
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nucleoprotein gene
also contains an important determinant of
virulence in trout, thought to be the result of
increased polymerase efficiency (Vakharia et al.
2019). The G protein performs the antigenic role
that stimulates host immune responses, and
genetic drift of this gene is expected to permit
evasion of developed immunity responses of
hosts (Panzarin et al., 2020; Vennerström et al.,
2020). The G gene has presented high genetic
diversity compared to other genes, and these
sequences have been widely used to evaluate
intraspecific phylogeography (Kuzmin et al.,
2009).
Recognizing how VHSV changes over time
in geographic locations and host species is
fundamental to predict future recurrence
patterns and enhance our understanding of rapid
evolution of pathogens (Domingo et al. 1998;
Stepien et al. 2015). This study examines the
hypothesis that VHSV genetic diversity is
heterogenous across years, sites, and host species
in the Thousand Islands region of the St.
Lawrence River, and that both physical and
biological structure of nursery bays influence the
viral genetic diversity achieved in different areas.
Methods
SPECIMEN COLLECTION
From 2018-2022, fish tissues were
preserved for viral testing from round gobies in
nursery bays of the Thousand Islands region
(Figure 1). In 2020-2021, tissue collection in these
sites was expanded to include potential native
reservoirs (i.e., yellow perch, rock bass, brown
bullhead, and Lepomis sunfish), and in 2022 a
VHSV-positive muskellunge was recovered from
the St. Lawrence River. A total of 1,520 fish were
screened for VHSV infection as part of Anna’s
dissertation research. From each fish, a pooled
sample of liver kidney and spleen along with a
separate sample of brain was collected and stored
in 200 µL of RNALater and frozen at -20°C.
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Viral RNA was
extracted from 50 µL
of homogenized tissue
supernatant using a
MagMax magnetic
bead extraction and a
MagMax-96 viral
isolation kit following
the protocols
described in the kit
and manufacturer’s
extraction program
AM1836_DW_50_V2.
Fish were tested for
VHSV using the qRT-
PCR assay described
by Hope et al. 2010,
detecting a ~300bp
fragment of the
nucleoprotein gene of
VHSV. Samples that
tested positive for
Figure 1 Map showing locations of nursery bays where wild fish were collected for this
study.
Esri, HERE, Garmin, (c)
OpenStreetMap contributors, and
the GIS user community
Garlock Bay
Cobb Sho~'Swan Bay
Flynn Bay Blind ~ay
o 0 Frinks Bay
Rose Bay
•
Waddington
0
Peas Bay • Millens Bay
0 12.5 25
Legend
Region
0 Alexandria Bay
• Cape Vincent
O Clayton - Grindstone
O Waddington
Ki lometers
50 Esri, HERE, Garmin, (c) OpenStreetMap contributors , and the GIS
user community
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VHSV infection with
a concentration exceeding 1x103 N-gene
copies/50 ng RNA were further pursued for
genetic sequencing.
REVERSE TRANSCRIPTION
Reverse transcription was completed
following the instructions in the SuperScript III
First-Strand Synthesis System (Invitrogen) using
random priming method. In each cDNA reaction,
0.5 μL cDNA Random Primer Mix (New England
Biolabs) and 0.5 μL dNTPs (10 mM) were added to
5 μL total RNA, incubated at 65°C for 5 min,
placed on ice and centrifuged briefly to collect
reaction components. Then, 5 μL of cDNA
synthesis mix (1 μL 5× buffer, 2 μL MgCl2, 1 μL
DTT, 0.5 μL RNase OUT, 0.5 μL SuperScript III
reverse transcriptase) was added and incubated
at 50°C for 50 min, 85°C for 5 min, and placed on
ice. Lastly, 1 μL RNase H was added followed by
incubation at 37°C for 20 min to remove the
original viral RNA from the new synthesized
cDNA. A total of 11 μL of VHSV genome cDNA was
synthesized from the RNA extracted with the
MagMax-96 viral RNA isolation kit.
POLYMERASE CHAIN REACTION (PCR)
AND DNA PURIFICATION
Two amplicons were synthesized in 35
cycles of PCR using OneTaq HotStart DNA
Polymerase (New England Biolabs) and the
corresponding primer sets. Amplification was
conducted in a total volume of 20μL reactions
using a Thermo Forma Hybaid PxE PCR thermal
cycler. Reactions contained 1 μL cDNA, 4 μL of 5X
OneTaq Standard PCR Buffer, 0.4 μL dNTPs
(10mM), 0.1 μL OneTaq Hot Start Polymerase, 0.4
μL forward primer (10 μM), 0.4 μL reverse primer
(10 μM), and 13.7 μL nuclease-free water.
Amplicons were produced using the following
cycling program: 94°C for 30 s, followed by 35
cycles of 94°C for 25 s, 46°C for 45 s, 68°C for 1
min, with a final step of 68°C for 5 min.
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An 811 bp region of
the N gene was amplified using:
forward: 5′- GGGGACCCCAGACTGT-3’
reverse: 5′- TCTCTGTCACCTTGATCC-3’
A 914 bp region of the VHSV G gene was amplified
using:
forward: 5′- ACTACCTACACAGTGAC-3′
reverse: 5′-CAATTTGTCCCCGAATATCAT-3′
Amplicon quality was assessed by agarose
gel electrophoresis running 2 μL on a 1% agarose
gel. 15 μL of the remaining amplified DNA was
purified using the Exo-CIP Rapid PCR Cleanup Kit
(New England Biolabs) following manufacturer’s
recommendations. DNA concentration was
determined by fluorescence detection using a
Qubit® Fluorometer (Invitrogen) and the Quant-
iT™ dsDNA BR Assay kit (Life Technologies). The
resulting amplicons were sequenced on a Sanger
platform at the Biotechnology Resource Center,
Cornell University. The Glycoprotein fragment
was sequenced using the forward PCR primer,
and the nucleoprotein fragment was completed
with a primer that flanked the forward PCR
primer, with the sequence 3’-
GCAAGCACTGTCCGTACTTC-5’.
BIOINFORMATICS ANALYSIS
We define a haplotype as “a unique gene
sequence that differs by one or more nucleotide
substitutions” (Stepien et al. 2015). Sequence data
Figure 2 Annotated VHSV genome map displaying functional genes and trailer regions.
[ Nucleoprotein ]
3' UTR [Ph
VHSV
0 3000 6000 9000
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were trimmed,
aligned, checked, and edited manually using
Geneious Prime software version 2023.1.2. The
nucleoprotein consensus sequences were
trimmed to 717 nt, corresponding to nt 406 to 1,122
of the full genome. Glycoprotein consensus
sequences for each isolate were trimmed to 813
nt, corresponding to nt 3,348 to 4,160 of the full-
length genomic sequence. Isolates with identical
sequences were grouped together into ‘sequence
types’ so that each unique haplotype was
represented once in our trees. The 669 nt
representing the central G region, corresponding
to nt 3,409 to 4,077 of the full-length genomic
sequence will be given a ‘universal sequence
designator’ prior to publication. Amino acid
substitutions were evaluated for each haplotype,
with the Great Lakes index strain M103GL used as
a reference (GenBank accession no. GQ385941).
Multiple sequence alignments of the newly
detected VHSV central G sequence types and the
previously described sequences in the Laurentian
Great Lakes were performed using ClustalOmega
v.1.2.2. Phylogenetic analysis was conducted using
the MrBayes v.3.2.6 plugin in Geneious Prime
software. The analysis used the MCMC approach
and was run for 1,100,000 generations and
sampled every 200 cycles. The general-time-
reversible (GTR) substitution model was
employed. Tree drawing was performed using
Geneious Prime v.2023.1.2.
Results and
Discussion
SEQUENCE TYPES
93 nucleoprotein and 78 glycoprotein
sequences were characterized from the St.
Lawrence River in this study. Their collection
dates, sites, and host species are detailed in
appendix 1.
The partial nucleoprotein sequences
consisted of 13 unique sequence types with SNPs
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identified at 14
locations. All N variants differed from the index
strain by ≥3 nt changes (Figure 3). All
nucleoprotein isolates possessed a cytosine at
position 1057, and while this residue agrees with
the vcG001 type specimen MI03, it represents a
nonsynonymous substitution from the vcG002
reference strain originating from a St. Lawrence
River round goby collected in a 2006 outbreak
(GenBank accession no. KY359355.2).
Figure 3 Nucleoprotein genetic differences among 13 unique sequence types identified in the St. Lawrence River isolates.
(A) N Fragment represents the 714 bp N protein region sequenced, corresponding to nt 406-1,122 Of the 11,184 nt full-
length genome sequence of Great Lakes VHSV strain MI03. Small vertical lines indicate sites of genetic differences as a
composite including all variants, depicting synonymous and nonsynonymous as black or red, respectively. (B) Specific
sequence differences in each sequence type shown relative to MI03 as a reference sequence. Boxed nt indicate non-
synonymous changes that result in amino acid (AA) changes in predicted N protein sequence. For sites in which an AA
change occurred, the first letter shows AA in the MI03 sequence type and the second letter indicates AA in the variant
sequence type.
A)
N Fragment
400
VHSV
0
B)
600
3000
II
II
II
II
II
II
II
11
11
II
11
11
6000
Position
II
AA / Nonsynonymous / Synonymous
9000
Variant N 442 N 527 N 547 N 557 N 709 N 793 N 812 N 889 N 893 N 905 N 937 N 973 N 1024 N 1054 N 1057*
C
~
~
C
C
4 G C
A C
6 C C
7 A C
8 C
9 T C
10 ~ A C
11 C T C
12 ~ ~ C
13 A C
AA change ~ ~ ~ L-F*
No. nt diffferent No. of
from MI03 isolates
0 40
1 33
8
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Only four of
the described nucleoprotein haplotypes encoded
nonsynonymous substitutions, all generating
alanine to threonine amino acid changes. Three
variants contained one nonsynonymous change,
and one variant contained two AA changes. All
other variants only exhibited synonymous
substitutions and likely have the same functional
Figure 4 Glycoprotein genetic differences among 13 unique sequence types identified in the St. Lawrence River isolates.
(A) Horizontal line represents the 813 bp G protein region sequenced, corresponding to nt 3,409–4,077 Of the 11,184 nt
full-length genome sequence of Great Lakes VHSV strain MI03. Small vertical lines indicate sites of genetic differences
as a composite including all variants, and asterisks indicate at which genetic heterogeneity observed within single
isolates. (B) Specific sequence differences in each sequence type shown relative to MI03 as a reference sequence. Boxed
nt indicate non-synonymous changes that result in amino acid (AA) changes in predicted G protein sequence. For sites
in which an AA change occurred, the first letter shows AA in the MI03 sequence type and the second letter indicates
AA in the variant sequence type. Shaded columns and dotted lines represent overlap with the 669 nt central
glycoprotein partial sequence used to apply a universal sequence designation (USD).
VHSV
0 3000
Position
AA / Nonsynonymous / Synonymous
B)
No. nt diffferent No. of
G3359 G3397 G3536 G3557 G3650 G3671 G3698 G3746 G3816 G3829 G3849 G3887 G3890 G3989 G4007 G4061 G4111 G4154 fromMI03 isolates USD
---
A G A
G A G
G A G
G A
C G A G
G C A
G A G
A G A G
A C C A G I G A
G A G A G
A G A G
A C G A G
A C G A G
IK-E R-K 1-V
G
G
~ G
C G
~ G
C G
C G
~ G C
G
§ G C C
C G
C G
C
4 7 vcG047
5 2 vcG066
6 22 vcG076
6 12 Haws-4•
7 22 Haws-5*
7 2 Haws-6*
7 2 Haws-7*
7 4 Haws-8*
8 1 Haws-9•
8 vcG076
8 1 Haws- 11 *
8 1 Haws-12*
8 1 Haws-13*
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phenotype as the
type strain MI03. The most abundant haplotype in
these isolates had no nucleotide changes in
reference to MI03 based on the partial N
sequences. The second most abundant haplotype
had a single nonsynonymous change at nt 527
(Figure 3).
The partial glycoprotein sequences
contained 13 unique sequence types with SNPs
recorded at 18 locations (Figure 3). Among the 13
unique sequences, 12 central G types were
represented (Figure 4). Three of these vcG types
(vcG047, vcG066 and vcG076) were previously
detected in the St. Lawrence River, and nine of
these types have not been previously described
(See Appendix 2) (Cornwell et al. 2014, Getchell et
al. 2019). All isolates were different from the index
strain by ≤4 nucleotides, and every sequence
contained the C-G mutation at nt 4007 (cG pos.
599, appendix 2) that was first described to
distinguish vcG002. Two nonsynonymous
substitutions were found outside of the central G
region, both generating glycine to aspartic acid
changes. The G-A change at nt 3397 occurred in
every isolate, and this residue also matches the
vcG002 type strain. The AA change at nt 3359 only
occurred in a single isolate, and its central G
sequence corresponded to vcG079 (Figure 4).
Four nonsynonymous substitutions were
observed in the central G region at nt 3816, 3829,
3849, and 3989 (cG pos. 406, 421, 441, and 581,
Appendix 2). Each isolate had a G-A substitution
at position 3829, creating an Arginine to Lysine AA
change. This SNP was first described in vcG014 by
Cornwell et al. 2012. 9/13 sequence variants
experienced the AA change at nt 3849 first
identified in vcG047 (Cornwell et al. 2014), 6/13
had the nt 3989 change described in vcG070
(Getchell et al. 2019), and only one variant had the
nonsynonymous change at nt 3819, novel to this
study’s haplotype Haws-9. The two haplotypes
that were the most abundant sequence types in
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these isolates,
vcG076 and Haws-5, differ only by a single
synonymous change at nt 3671 (Figure 4).
PHYLOGENETIC ANALYSIS
Phylogenetic analysis of central G
sequence types observed in this study displays
expanding genetic diversity in the St. Lawrence
River, descending from variants previously
described in genetic sequencing efforts.
Relationships in the central G tree show the
sequence types detected in this study were most
closely grouped to vcG014, described by Cornwell
et al. 2012. (Figure 5).
SPATIAL ASSOCIATIONS
Multiple sequence types were found in
each sub-region of the USLR. The primary 3
regions possessed similar VHSV G-gene
richness, with seven haplotypes observed in the
Cape Vincent and Alexandria Bay isolates, and six
in the Clayton-Grindstone samples (Figure 6A).
Six of the G-gene haplotypes only
occurred in one of the geographic regions with
an ephemeral occurrence in 1-2 isolates (Haws-
6, 7, 9, 11, 12, and 13). Four haplotypes (vcG076,
Haws-4, Haws-5, Haws-8) occurred across the
three sub-regions of the study area, and they all
also occurred in multiple regions simultaneously
during a single sampling year.
Three previously described sequence
types identified by Cornwell et al. 2014 (vcG047),
and Getchell et al. 2019 (vcG069 and vcG076),
appeared among the isolates in this study.
vcG076 was one of the most abundant sequence
types across geographic regions, but both
vcG069 and vcG047 were only detected again in
isolated incidents. vcG069 was both described
and reobserved in Cape Vincent, while vcG066
was initially described from Clayton in 2017, but
found in Alexandria Bay thereafter.
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Figure 5. Phylogenetic relationships among the St. Lawrence River 2019-2022 sequence types and the previously
described partial glycoprotein (G) sequence types (669 nt) from the Laurentian Great Lakes. Labels for all
branches indicate individual virus isolate designations. Labels with an asterisk indicates sequences reported
in this study. Posterior probability values are shown at the branch nodes and the scale bar indicates the number
of substitutions per nt site.
0.95
vcG008
vcG011
vcG010
vcG007
vcG006
vcG00S
vcG004
vcG003
--------------------ovcG009
vcG051
vcG0S0
vcG048
vcG045
vcG032
vcG016
vcG015
vcG002
0.9
vcG017
0.93
vcG018
vcG014
0.9
0.96
vcG049
vcG046
--------------------0 Haws-9*
vcG047*
0.65
0.51
.-----o vcG066*
--------o Haws-8*
L-----..;;
0;.;..·7..;;9~-------o Haws-5*
vcG076*
-------a Haws-13*
1--------0 Haws-12*
L_ ________ ....:0::.:.-:::.:98:.v-----o Haws-1 1 *
Haws-7*
Haws-4*
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Spatial
trends display the connectivity of the St.
Lawrence ecosystem, and that the VHSV genetic
structure contains both geographically
widespread and locally isolated sequences.
Sequence types that are sustained across
multiple seasons are capable of persisting in the
same location and dispersing throughout the
ecosystem.
YEAR ASSOCIATIONS
Several central G haplotypes were
detected during each of the sampling years.
Among the most dominant haplotypes, vcG079
presented continuous presence during every
sampling year between 2017-2022, but the
relative proportion of this haplotype declined
between 2021-2022. Haws-5 was first identified
among the 2020 isolates and increased in
relative proportion until 2022. Haws-4 also
appeared in the 2020 isolates and maintained
similar relative proportions throughout the study
period. Haws-8 occurred in 2021 isolates and
was observed again in 2022 (Figure 6). vcG047
was first described in samples collected during
2011 (Cornwell et al. 2014), and was reobserved in
2017, and again 2022. vcG066 was first observed
in 2017 samples (Getchell et al. 2019) and
appeared again in a single isolate from 2020.
Ephemeral haplotypes were also observed during
each of the study years. 2020 presented three
unique haplotypes, 2021 had one, and 2022 had
two.
This data shows that genetic haplotypes
are capable of sustaining infections in the St.
Lawrence River for extended periods of time,
with the oldest detected type dated back to at
least 2011. Each outbreak season also produced
new genetic variation, and only a limited amount
of the overall diversity observed persisted
between seasons.
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Figure 6. Distribution of identified VHSV central G sequence types isolated among (A) different sub-
regions in the upper St. Lawrence River, (B) different isolation years, and (C) different host species.
A
Cape Vincent
C
_o Clayton - Grindstone
+-'
ro
(.)
Alexandria Bay 0
_J
Waddington I USO
0 10 20 30 40 ■ vcG047
B ■ vcG066
2022 ■ vcG076
■ Haws-4
2021
■ Haws-5 s....
ro
2020 ■ ~ Haws-6
2019 I ■ Haws-7
2017 ■ ■ Haws-8
■ Haws-9
0 10 20 30 40 ■ Haws-11
C ■ Haws-12
CJ) Muskellunge I ■ Haws-13
(l)
·u
Lepomis sp. (l)
a.
CJ)
+-' Yellow perch
CJ)
0
I Round goby
0 20 40 60
No. of isolates
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~ ~ RESOURCES INSTITUTE
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This report was prepared for the New York State Water Resources Institute (NYSWRI) and the
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HOST SPECIES ASSOCIATIONS
Round goby sequences comprised the
majority of isolates that were successfully
sequenced in this study. All six of the unique
ephemeral sequences occurred in a round goby
host, and all sequences found in other host
species were also detected in round gobies. Two
of the previously described haplotypes, vcG047
and vcG069, were also only observed in round
gobies.
For 21 individual hosts, both the brain and
pooled organ samples were amplified and
sequenced, and 7/21 of these fish possessed
different haplotypes between the brain and
pooled organ isolations. Sequence heterogeneity
was observed in infected round gobies,
pumpkinseed, and muskellunge. The muskie
mortality contained a mixture of the two most
abundant haplotypes, vcG079 in its pooled organ
sample, and Haws-5 in the brain.
Round gobies both sustain abundant and
widespread haplotypes and generate new genetic
variation. Our data suggests that native hosts are
exposed to the genetic structure contained in
round goby infections, and that the most
abundant haplotypes are likely to be those
present in infected native fishes.
Conclusions
Increased surveillance for VHSV in free-
ranging fish and active assessment of risk factors
to prevent further virus impacts are major
components of the action items promoted by
USDA-APHIS in the Laurentian Great Lakes
(Gustafson et al. 2018; Thompson et al. 2011).
Routine and spatially robust genetic typing of
field isolates is necessary to track the genetic
diversity and epidemiology of VHSV through
evolutionary time, across the basin. Isolates with
the same genetic sequences are most likely
epidemiologically linked, and therefore using
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SNPs as genetic tags
is useful to investigate viral movement and
evolutionary patterns (Thompson et al. 2011).
This research shows the widespread and
continued presence of VHSV in the upper St.
Lawrence River and highlights the continued
need to prevent the spread of new VHSV variants
into additional water bodies. The genetic
composition of VHSV that exists in the St.
Lawrence River is substantially diverged from the
original reference strain, illustrating this virus’
capacity to exploit mutation to persist in the
environment. Monitoring the viral composition of
round gobies provides a comprehensive
illustration of the genetic diversity present in the
overall fish community at a location and time.
Student Training
One Ph.D. level graduate student, Mrs. Anna
Haws, was trained in genetic sequence
preparation and analysis during this project. She
conducted all reverse transcription and
amplification reactions, PCR product cleaning,
DNA quantity/quality evaluation, and genetic
data preparation and analysis. This work will be
the final chapter in her doctoral dissertation for a
degree in Fish & Wildlife Biology & Management
from SUNY ESF.
Anna in the field holding a round goby
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This report was prepared for the New York State Water Resources Institute (NYSWRI) and the
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Publications/
Presentations
Haws, A.L., Getchell, R.G., Farrell, J.M. (2023). Viral
Hemorrhagic Septicemia Virus genetic diversity
in the Upper St. Lawrence River. Manuscript in
preparation.
Haws, A.L. “INTRODUCTIONS, INTERACTIONS,
AND IMPACTS: ROUND GOBY AND VIRAL
HEMORRHAGIC SPETICEMIA VIRUS IN THE
UPPER ST. LAWRENCE RIVER”. PhD dissertation,
SUNY College of Environmental Science and
Forestry, in preparation.
Haws, A.L., Getchell, R.G., Farrell, J.M. (July 24-27
2023). Influence of Round Gobies on VHSV
Disease Ecology in the Upper St. Lawrence River.
[Conference presentation]. 2023 meeting of the
AFS Fish Health Section.
Haws, A.L., Getchell, R.G., Farrell, J.M. (September
24-26). Influence of Round Gobies on VHSV
Disease Ecology in the Upper St. Lawrence River.
[Conference presentation]. 2023 NYS Invasive
Species Expo.
Haws, A.L., Getchell, R.G., Farrell, J.M. (TBD). Viral
Hemorrhagic Septicemia Virus genetic diversity
in the Upper St. Lawrence River. [Conference
presentation]. 2024 meeting of the New York
chapter of the American Fisheries Society.
References
Al-Hussinee, L., Lord, S., Stevenson, R., Casey, R.,
Groocock, G., Britt, K., Kohler, K., Wooster, G.,
Getchell, R., Bowser, P., & Lumsden, J. (2011).
Immunohistochemistry and pathology of multiple
Great Lakes fish from mortality events associated with
viral hemorrhagic septicemia virus type IVb. Diseases
of Aquatic Organisms, 93(2), 117–127.
Boonthai, T., Loch, T. P., Zhang, Q., Van Deuren, M. G.,
Faisal, M., Whelan, G. E., & Herbst, S. J. (2018). Retail
Baitfish in Michigan Harbor Serious Fish Viral
Pathogens. Journal of Aquatic Animal Health, 30(4),
253–263.
Cornwell, E. R., Eckerlin, G. E., Thompson, T. M., Batts,
W. N., Getchell, R. G., Groocock, G. H., Bowser, P. R.
(2012). Predictive factors and viral genetic diversity for
viral hemorrhagic septicemia virus infection in Lake
Ontario and the St. Lawrence River. Journal of Great
Lakes Research, 38(2), 278–288.
Cornwell, E. R., Primus, A., Wong, P. T., Anderson, G. B.,
Thompson, T. M., Kurath, G., Getchell, R. G. (2014).
Round gobies are an important part of VHS genotype
IVb ecology in the St. Lawrence River and eastern Lake
Ontario. Journal of Great Lakes Research, 40(4), 1002–
1009.
Domingo, E. (1998). Quasispecies Structure and
Persistence of RNA Viruses. Emerging Infectious
Diseases, 4(4), 521–527.
Eckerlin, G. E., Farrell, J. M., Casey, R. N., Hope, K. M.,
Groocock, G. H., Bowser, P. R., & Casey, J. (2011).
Temporal Variation in Prevalence of Viral Hemorrhagic
Septicemia Virus Type IVb among Upper St. Lawrence
River Smallmouth Bass. Transactions of the American
Fisheries Society, 140(3), 529–536.
Farrell, J. Getchell, R., Kapuscinski, K., LaPan, S. (2017).
Long-Term Trends of St. Lawrence River Muskellunge:
Effects of Viral Hemorrhagic Septicemia and Round
Goby Proliferation Creates Uncertainty for Population
Sustainability. American Fisheries Society Symposium.
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NYS DEC Bureau of Invasive Species and Ecosystem Health.
Getchell, R. G., E. J.,
Bogdanowicz, S. M., Andrés, J. A., Schulman, A. T.,
Kramer, J., Marquis, H. (2019). Investigation of round
goby viral haemorrhagic septicaemia outbreak in New
York. Journal of Fish Diseases, 42(7), 1023–1033.
Gustafson, L. L., Remmenga, M. D., Gardner, I. A.,
Hartman, K. H., Creekmore, L. H., Goodwin, A. E.,
Whaley, J. E., Warg, J. V., Gardner, S. L., & Scott, A. E.
(2014). Viral hemorrhagic septicemia IVb status in the
United States: Inferences from surveillance activities
and regional context. Preventive Veterinary Medicine,
114(3–4),
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of diagnostic test for aquatic animals: chapter 2.3.10
viral haemorrhagic septicaemia. Edited by World
Organization of health.
Stepien, C. A., Pierce, L. R., Leaman, D. W., Niner, M. D.,
Shepherd, B. S., & Chen, T. Y. (2015). Gene
diversification of an emerging pathogen: A decade of
mutation in a novel fish Viral Hemorrhagic Septicemia
(VHS) substrain since its first appearance in the
Laurentian Great Lakes. PLoS ONE, 10(8), 1–26.
Stepien, C. A., & Niner, M. D. (2020). Evolutionary
trajectory of fish Piscine novirhabdovirus (=Viral
Hemorrhagic Septicemia Virus) across its Laurentian
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Appendices
Appendix 1. Summary of isolates used in this study
and sequences obtained.
Appendix 2. Summary of previously described USD
variants in Laurentian Great Lakes used in
phylogenetic analyses.
Appendix 3. Methods addendum.
Disclaimer
The views and conclusions contained in this document
are those of the authors and should not be interpreted as
representing the opinions or policies of the funding
organizations or agencies. Mention of trade names or
commercial products does not constitute their
endorsement by these organizations.
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Appendix 1. Summary of isolates
used in this study and sequences
obtained.
17-GETCHELL
TIBS1
17-GETCHELL·
TIBS3
17-GETCHELL
TIBSS
17-GETCHELL
TIB57
19-3B
19-13P
19-16P
19-18B
20-459P
20-4UP
20-471P
20-652P
20-826B
20-843P
20-888P
20·889P
20-893-P
20-898B
20-902B
20-902P
Dato
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Rose Bay 5/>