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dc.contributor.authorPatashnick, Susanne
dc.date.accessioned2009-10-23T15:21:25Z
dc.date.available2009-10-23T15:21:25Z
dc.date.issued2009-10-28
dc.identifier.urihttps://hdl.handle.net/1813/14129
dc.description.abstractA six year old female spayed Pomeranian dog presented to Cornell with vestibular signs localizable to the cerebellum. MRI identified a well-demarcated, wedge-shaped lesion in the distribution of the right rostral cerebellar artery which was hyperintense on T2 and mildly contrast-enhancing, a classic presentation for vascular accident. Cerebrospinal fluid analysis was unremarkable. Ancillary testing revealed azotemia, mild nonregenerative anemia, increased urine protein-to-creatinine ratio, increased fibrinogen, increased D-dimers, and decreased antithrombin. Based on the results of these tests, a diagnosis of cerebellar vascular accident secondary to protein losing nephropathy was made. Cerebellar vascular accident pathophysiology, clinical signs, diagnostics, imaging, underlying causes, treatment, and prognosis are briefly discussed.
dc.language.isoen_USen_US
dc.relation.ispartofseriesSenior seminar paper
dc.relation.ispartofseriesSeminar SF610.1 2010
dc.subjectDogs -- Diseases -- Case studiesen_US
dc.titleCerebellar vascular accident secondary to protein losing nephropathy in a Pomeranianen_US
dc.typeterm paperen_US


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