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dc.contributor.authorWalker, Melissa
dc.date.accessioned2009-09-17T18:31:38Z
dc.date.available2009-09-17T18:31:38Z
dc.date.issued2003-10-10
dc.identifier.urihttps://hdl.handle.net/1813/13690
dc.description.abstractA 2 year old, castrated male West Highland White Terrier was referred for evaluation of vomiting, anorexia, polyuria, polydipsia, and lethargy. Copper storage disease and fanconi's syndrome was diagnosed via quantitative liver copper analysis, copper stains, and routine histological evaluation of a laparoscopic liver sample. Fanconi's syndrome was diagnosed based on an increased fractional excretion of amino acids, organic acids, and electrolytes, and glucose in the face of euglycemia. The dog received supportive therapy and was treated with D-penicillamine, ursodiol, vitamin E, S-adenosylmethionine. At four-month recheck he was clinically normal and the fanconi's syndrome has resolved.en_US
dc.language.isoen_USen_US
dc.relation.ispartofseriesSenior seminar paper
dc.relation.ispartofseriesSeminar SF610.1 2004 W35
dc.subjectDogs -- Diseases -- Case studiesen_US
dc.titleCopper storage disease and Fanconi's syndrome in a West Highland White Terrieren_US
dc.typeterm paperen_US


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